There is disagreement about the

There is disagreement about the pathogenesis of SID. Irving and Lister, in explaining a correlation between SID and omphalocele, suggested that temporary extrinsic Dioscin manufacturer and bowel entrapment could lead to SID. However, Heller and Waag noted that SID can develop even in the bowel that remains within the abdomen and would not be subjected to extrinsic compression. Some authors have called SID a giant Meckel diverticulum because the two entities have several similar characteristics. SID may contain heterotopic gastric or pancreatic tissue and is commonly located in the ileum, although any sites throughout the digestive tract may be involved. Furthermore, an apparent feeding vessel to SID originating from the mesentery resembles the vitelline vessel seen with the Meckel diverticulum. Heller and Waag suggested that the failure of the notochord to separate from the endoderm could account for either a Meckel diverticulum or SID. Definitive treatment for SID is resection with end-to-end anastomosis and is recommended even if the patient remains asymptomatic. However, when surgery for omphalocele is also required, there is a question as to whether increased intra-abdominal pressure following primary abdominal closure for omphalocele increases the risk of leakage of bowel anastomosis or not. Basaran and colleagues suggested that a decrease in abdominal content after the dilated bowel was resected would make abdominal closure easier. In the published cases of patients with omphalocele and SID treated with primary resection and abdominal closure in a single operative setting, there were no reports of postoperative anastomotic leakage. Thus, Transformation seems feasible to simultaneously treat SID and omphalocele. As our case demonstrates, interval resection of the involved bowel is an alternative, although one may doubt if it is necessary to subject an infant to two separate procedures.
Conclusion